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Soft Tissue Recession Around Implants – A common Complication

Blog English, Implantology

Introduction

One of the major concerns is soft tissue recession around implants thay may appear over time (Adell et al. 1981; Grunder 2000) and this is an important issue not only for the patient but also for the clinician.

Today one of the first considerations for implant therapy is to achieve aesthetics results that are in harmony with the surrounding tissues.

Although this is a very familiar finding in our daily practice, literature is lacking regarding approaches to solve this problem of recessions involving implants predictably (Roccuzzo et al. 2002; Mareque-Bueno 2011; Zucchelli et al.2012).

Soft Tissue Recession Around Implants

A 62 years old lady attended for a review appointment, her main complaint was the recession around an implant in the #14 implant position. The patient is fit and healthy and a non smoker. Lets consider the causes that could lead to this recession. I scored the following causes with 0 if it is very improbable to be the cause of the recession, 1 maybe and 2 whereas it is probably the main cause:

  1. Mucosal quality (keratinized vs. Non-keratinized)

This is improbable to be the main cause. The amount of keratinized tissue is more than 1 mm and this is unlikely to be a cause so the score for this one is 0.

  1. Mucosal attachment (mobile vs. Non-mobile)

There was enough attached mucosa and we should discard this cause. Score 0.

  1. Mucosal thickness

Very thin mucosa was found. This is probably one of the main causes. Score 2.

  1. Facial bone crest level and thickness

During the peri-implnt probing depth (PPD) at buccal aspect was +/- 6 mm so we should give this a score 2.

  1. Implant fixture angle

The angulation of the abutment was correct from the prosthetic and biological point of view. Score 0.

  1. Interproximal bone crest level

Both interproximal peaks of bone crest were well preserved. Score 0.

  1. Level of first bone to implant contact

As I previously mentioned, the depth on probing was 6 mm. Score 2.

  1. Depth of Implant platform

The platform depth was above the bone crest. Score 0.

  1. Micro and macrostructure of the implant neck.

The microstructure are microthreads and would not influence soft tissue recession. Score 0.

  1. Implant-abutment and prosthesis connection

I used a intermediate abutment (Aurea, Phibo, Spain), during the impressions to avoid recession caused by the connection and disconnection of the abutments. Score 0.

  1. Surgical technique

The surgical procedure was uneventful. 2 years ago a sinus floor augmentation was perfomed allowed to consolidate before implants were placed. Score 0.

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Fig. 1. Soft issue recession around an implant 1 year after loading.

The total score was 6 out of 22 where over 10 we should consider that maybe the treatment option should be to remove the implant and site form the area to avoid recession????

After reviewing all this point I had made a decision based where the score was 2. So we have thin facial bone crest, thin mucosa and the level of the first bone to implant contact was low so we had to solve two points.

  1. Bone thickness and first implant bone contact: The approach here should involve GBR with a bone sustitution and a membrane
  2. Thin soft tissue: At this pointg the only option was to perform a soft tissue augmentation.

Considering the two approaches described previously, I decided to perform a guided bone regeneration simultaneously to the soft tissue augmentaation.

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Fig. 2. Removing the fixed prosthesis is mandatory in this case to control the soft tissue management.

Surgery to solve soft tissue recession 

The first step was to remove the fixed prothesis. This will allow us to perform a coronally advanced flap and will facilitate both adavanced coronally flap surgery and connective tissue harvesting surgery from the palatal.

First connective tissue graft was harvested from the palatal. 4/0 PTFE sutures were used to suture the wound.

A flap with beveled distal and mesial vertical releasing incisions were raised avoiding the canine and the adjacent implant. Several incisions were performed apically in the periosteum so the flap can be mobilised coronally and cover the recession.

GBR was performed utilising Bio-Oss (Geistlich, Switzerland) mixed the autologous bone (50/50) and a collagen membrane Creos (Nobel Biocare). The membrane was cut in two pieces and used to cover the biomaterial, one was placed vertically and the other horizontally so it can stabilise the biomaterial.

The connective tissue was then sutured to the implant neck with 5/0 monofilament sutures and covered with the flap.

Antibiotic amoxicilin 500 mg and anti-inflammatories were prescribed to the patient during 5 days.

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Fig. 2-3-4. Connective tissue graft harvested from the palatal and on the right photo the coronally advanced flap and sutured. The provisional caps can also be seen attached to the abutments of the adjacent implants. Occlusal view it can be observed the donor site sutured with 4/0 PTFE sutures.

Conclusion

After 15 days of healing the patient came to the office and an improvement in the soft tissue level could be observed. Long term observation is needed to confirm the improvement of the soft tissue thickness and the partial coverage of the recession.

Although soft tissue management around peri-implant tissues are not always predictable, but as in teeth some approaches can be performed to solve soft tissue recessions around implants such as a coronally repositioned flap with simultaneous connective tissue graft.

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Fig. 5. Final result after 15 days.

 

 

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